Toxic pollutants contribute to obesity

Environmental toxins – external toxins that harm through contamination of drinking water, foodstuffs, or exposure resulting in inhalation or penetration through the skin are numerous and vary according to the geographical latitude and social development of individual countries. Their most common source is power plants, non-ferrous metal plants, mines, and wells, as well as pesticides, herbicides, and polluted air. Internal toxins accumulate mainly in the abdominal organs as a result of metabolizing products and substances taken with food or breathing.

In a normal and balanced environment that chronologically preceded the industrial revolution in Europe, most people, or at least those of mature age, were able to maintain their health, thanks to their endogenous detoxification mechanisms. But in the modern world, the environment is so polluted that the abilities of the human organism are severely outnumbered by countless pollutants. Our inability to shake them off allows them to be stored in fatty tissues.

When a person loses weight, an amount of fat is lost, in which there are stored toxins, not removed from the body. Such toxins are DDT, bisphenol, and others. They break free from their fat prisons and enter the bloodstream, disrupting the body’s natural metabolism. It has been suggested that such toxins can seriously slow down fat loss regardless of regular exercise and a proper diet
feeding.

Dr. Sheila Dean and her team found in 2007 that toxins alter metabolism, disrupt hormone function, damage cell mitochondria, and increase oxidative stress. A decrease in thyroid hormones and a change in circadian rhythms are also caused by similar internal poisonings.

According to a study published in the respected Italian Journal of Pediatrics, the toxin bisphenol A is among the pollutants that cause obesity through hyperlipidemia – elevated levels of lipids in the blood caused by hormonal imbalance due to intoxication with bisphenol A. It also inhibits the transport of glucose in fat cells and suppresses the release of adipokine – an immunomodulatory compound important for normal fat metabolism.